Abstract
The Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue growth. It is an evolutionarily conserved signaling cascade regulating numerous biological processes, including cell growth and fate decision, organ size ...Read More
Keywords
Drosophila melanogasterRegulatorCell biologyCell fate determinationBiologyMacrophageCholesterolMetabolismCell growthSignal transductionBiochemistryChemistryTranscription factorGene
Related Publications
STRUCTURES AND FUNCTIONS OF MULTILIGAND LIPOPROTEIN RECEPTORS: Macrophage Scavenger Receptors and LDL Receptor-Related Protein (LRP)
The Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue growth. It is an evolutionarily conserved signaling cascade regulating numerou...
THE HEAT-SHOCK RESPONSE
The Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue growth. It is an evolutionarily conserved signaling cascade regulating numerou...
<i>ras</i> GENES
The Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue growth. It is an evolutionarily conserved signaling cascade regulating numerou...
THE FAMILY OF COLLAGEN GENES
The Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue growth. It is an evolutionarily conserved signaling cascade regulating numerou...
DNA METHYLATION AND GENE ACTIVITY
The Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue growth. It is an evolutionarily conserved signaling cascade regulating numerou...
Publication Info
- Year
- 1983
- Type
- review
- Volume
- 52
- Issue
- 1
- Pages
- 223-261
- Citations
- 2302
- Access
- Closed
External Links
Social Impact
Altmetric
PlumX Metrics
Social media, news, blog, policy document mentions
Citation Metrics
2302
OpenAlex
Cite This
Michael S. Brown,
Joseph L. Goldstein
(1983).
LIPOPROTEIN METABOLISM IN THE MACROPHAGE: Implications for Cholesterol Deposition in Atherosclerosis.
Annual Review of Biochemistry
, 52
(1)
, 223-261.
https://doi.org/10.1146/annurev.bi.52.070183.001255
Identifiers
- DOI
- 10.1146/annurev.bi.52.070183.001255